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The calcium sulfate in a desiccator is effective at removing water vapor only as long as some anhydrous salt remains. E-print hep-th0403166. Nonsurgical strategies, 377-380 techniques, 383 Oropharyngeal tumors lymph node management, 321 positive for HPV, 161 and tobacco smoke, 214-216 Oropharynx, preservation, 478-489 OSI-774 (Tarceva), 539-540 Outcome assessment cost of care, 441 health status, 436-438 historical background, 433-434 morbidity, 436 quality of free binary option full Tuvalu, 438-441 survival, 434 actuarial, 435-436 determinate, 434-435 Kaplan-Meier, 436 Overexpression bFGF, 87 cyclin D1, 109 FAK, 70 oncogenes, 117 p53, 124, 529-530 serine proteases, 140 Oxaliplatin, 537 Oxidative stress, tobacco and alcohol supraglottic, 386-387 vertical, 384-385 Passive immunotherapy, 577 Pathology ACC, 16-17 anaplastic thyroid carcinoma, 418 follicular thyroid carcinoma, 415 HNSCC, 81-82 Htirthle cell tumor, 416 medullary thyroid carcinoma, 417 nasopharyngeal carcinomas, 492--493 papillary thyroid carcinoma, 413 skull base-associated, 342-346 PD-ECGF, expression in HNSCC, 90 P15-E, detection in HNSCC, 575 Performance status scale, head and neck cancer patients, 437-438 PET detection of recurrent disease, 31-32 with FDG, s e e FDG PET staging, 27-28 Pharmacologic interventions, smoking cessation, 193-195 Pharynx, s e e a l s o Oropharynx cancer, 317 normal anatomy, 36 Phenotypic changes, associated with field carcinogenesis, 236 Phonomicrosurgical approach, T1glottic cancer treatment, 360 Phosphoinositide-3 OH kinase, s e e PI3K Photon radiation, for ACC, 19-20 Physician-based intervention, smoking cessation, 192-194 Physiological monitoring, during skull base surgery, 342 Phytochemical supplementation, 207-208 PI3K, in tumor cell invasion, 71 Plakoglobin, Armadillo repeats, 71-72 Plasminogen activation system, 137-138 Platelet-derived endothelial cell growth factor, s e e PD-ECGF Platinum agents chemotherapy based on, 332 Nedaplatin, 537 Ploidy, cytometric determination, 168-169 Polymerase chain reaction, detection of HPVs, 155-161 Polymers, gene therapy based on, 559 Polymorphisms in cell cycle genes, 220-221 in DNA repair genes, 219-220 p21WAF1, 122 Positron emission tomography, s e e PET PRAD- 1, cell signaling, 119 Preauricular infratemporal fossa, dissection, 350-352 Premature chromosome condensation, 232 Primary carcinoma clonal origin, 276 unknown, metastatic to cervical nodes, 9-13 Prognosis follicular thyroid carcinoma, 415-416 medullary thyroid carcinoma, 417 papillary carcinomas, 413-414 p12D~ pl6 as CKI, 108 effects, 203 Index 5 9 9 CDKN2-encoding, 122-123 inactivation, 106-107 interaction with high-risk HPV E6 protein, 153-154 p 16Ink4A, loss, 109 p 18INK4c,108 p21 expression independent of p53, 104 inhibition of DNA replication, 105 p21WAF1, 109, 122, 202 p27, role in programmed cell death, 105-106 p4442MAPK,upregulation, 87 p53 Ad5CMVp53, 546 as biomarker for predicting cancer, 296-297 CP.

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